Moreau, R; Clària, J; Aguilar, F; Fenaille, F; Lozano, JJ; Junot, C; Colsch, B; Caraceni, P; Trebicka, J; Pavesi, M; Alessandria, C; Nevens, F; Saliba, F; Welzel, TM; Albillos, A; Gustot, T; Fernández, J; Moreno, C; Baldassarre, M; Zaccherini, G; Piano, S; Montagnese, S; Vargas, V; Genescà, J; Solà, E; Bernal, W; Butin, N; Hautbergue, T; Cholet, S; Castelli, F; Jansen, C; Steib, C; Campion, D; Mookerjee, R; Rodríguez-Gandía, M; Soriano, G; Durand, F; Benten, D; Bañares, R; Stauber, RE; Gronbaek, H; Coenraad, MJ; Ginès, P; Gerbes, A; Jalan, R; Bernardi, M; Arroyo, V; Angeli, P; CANONIC Study Investigators of the EASL Clif Consortium; Grifols Chair and the European Foundation for the Study of Chronic Liver Failure (EF Clif).
Correction to 'Blood metabolomics uncovers inflammation-associated mitochondrial dysfunction as a potential mechanism underlying ACLF' [J Hepatol 2020 (72) 688-701].
J Hepatol. 2020; 72(6):1218-1220
Doi: 10.1016/j.jhep.2020.03.002
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