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Reindl, O; Loidl, A; Franz, B; Hofer, JF; Pichler, R.
Pitfall in follow-up imaging of pancreatic neuroendocrine tumor by somatostatin receptor PET.
Neuro Endocrinol Lett. 2013; 34(4):273-274
(- Case Report)
Web of Science
PubMed
- Co-Autor*innen der Med Uni Graz
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Pichler Robert
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- Abstract:
- 56-year old woman was operated of a pancreatic NET in May 2011. Abdominal pain had led to imaging and consecutively the finding of cholecystolithiasis and the tumor. The gall bladder, left hemi-pancreas, regional lymph nodes and the (unintentional injured) spleen were resected. At routine control examination in October 2012 CT presented three contract enhancing intra-abdominal lesions with a diameter of 2-3.5 cm. Consecutively 68Ga-DOTA-NOC PET-CT showed high tracer uptake (SUV 10-12) at these lesions. Therefore a relapse of the neuro-endocrine tumor was suspected. After reoperation in December 2012 histology did not reveal any sign of neuroendocrine tumor but identified spleen tissue most probably caused by splenosis accidentally seeded at the first operation. Physiologically the spleen is highly avid at 68Ga-DOTATOC PET, but splenosis presents with less standard uptake value. In our case the described lesions presented with an SUV quite comparable to that of neuroendocrine tumor tissue.
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Diagnosis, Differential -
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Female -
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Humans -
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Middle Aged -
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Neoplasm Recurrence, Local - metabolism
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Neuroendocrine Tumors - metabolism
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Organometallic Compounds - diagnostic use
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Pancreatic Neoplasms - metabolism
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Positron-Emission Tomography -
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Radiopharmaceuticals - diagnostic use
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Receptors, Somatostatin - metabolism
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Splenosis - metabolism
- Find related publications in this database (Keywords)
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neuroendocrine tumor
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somatostatin receptor
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follow up imaging
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PET