** = Publikationen gelistet in SCI/SSCI/Pubmed
** Schulz, E; Lind, K; Renner, W; Petersen, BS; Quehenberger, F; Dill, C; Hofer, S; Lal, R; Hoefler, G; Schlenke, P; Ehninger, G; Schetelig, J; Middeke, JM; Stölzel, F; Sill, H
The TP53 Pro72Arg SNP in de novo acute myeloid leukaemia - results of two cohort studies involving 215 patients and 3759 controls.
Br J Haematol. 2018; 181(1):148-151
Doi: 10.1111/bjh.14527
[OPEN ACCESS]
Web of Science
PubMed
FullText
FullText_MUG
** Lal, R; Lind, K; Heitzer, E; Ulz, P; Aubell, K; Kashofer, K; Middeke, JM; Thiede, C; Schulz, E; Rosenberger, A; Hofer, S; Feilhauer, B; Rinner, B; Svendova, V; Schimek, MG; Rücker, FG; Hoefler, G; Döhner, K; Zebisch, A; Wölfler, A; Sill, H
Somatic TP53 mutations characterize preleukemic stem cells in acute myeloid leukemia.
Blood. 2017; 129(18): 2587-2591.
Doi: 10.1182/blood-2016-11-751008
[OPEN ACCESS]
Web of Science
PubMed
FullText
FullText_MUG
** Zebisch, A; Lal, R; Müller, M; Lind, K; Kashofer, K; Girschikofsky, M; Fuchs, D; Wölfler, A; Geigl, JB; Sill, H
Acute myeloid leukemia with TP53 germ line mutations.
Blood. 2016; 128(18): 2270-2272.
Doi: 10.1182/blood-2016-08-732610
[OPEN ACCESS]
Web of Science
PubMed
FullText
FullText_MUG
** Schulz, E; Lind, K; Renner, W; Petersen, BS; Dill, C; Hofer, S; Lal, R; Quehenberger, F; Stolzel, F; Sill, H
THE HOMOZYGOUS TP53 P72R SNP IS ASSOCIATED WITH NON-FAVORABLE CYTOGENETIC RISK GROUPS IN ACUTE MYELOID LEUKEMIA
HAEMATOLOGICA. 2016; 101: 383-383.
[Poster]
Web of Science
** Zebisch, A; Lal, R; Muller, M; Lind, K; Sill, H
TP53 germline mutations occur in acute myeloid leukemia and are associated with therapy-related cases following ionizing radiation
WIEN KLIN WOCHENSCHR. 2016; 128: S385-S386.
[Poster]
Web of Science