** = Publikationen gelistet in SCI/SSCI/Pubmed
** Dieudonné, Y; Lorenzetti, R; Rottura, J; Janowska, I; Frenger, Q; Jacquel, L; Vollmer, O; Carbone, F; Chengsong, Z; Luka, M; Depauw, S; Wadier, N; Giorgiutti, S; Nespola, B; Herb, A; Voll, RE; Guffroy, A; Poindron, V; Ménager, M; Martin, T; Soulas-Sprauel, P; Rizzi, M; Korganow, AS; Gies, V
Defective germinal center selection results in persistence of self-reactive B cells from the primary to the secondary repertoire in Primary Antiphospholipid Syndrome.
Nat Commun. 2024; 15(1): 9921
Doi: 10.1038/s41467-024-54228-8
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** Staniek, J; Kalina, T; Andrieux, G; Boerries, M; Janowska, I; Fuentes, M; Díez, P; Bakardjieva, M; Stancikova, J; Raabe, J; Neumann, J; Schwenk, S; Arpesella, L; Stuchly, J; Benes, V; García, Valiente, R; Fernández, García, J; Carsetti, R; Piano, Mortari, E; Catala, A; de, la, Calle, O; Sogkas, G; Neven, B; Rieux-Laucat, F; Magerus, A; Neth, O; Olbrich, P; Voll, RE; Alsina, L; Allende, LM; Gonzalez-Granado, LI; Böhler, C; Thiel, J; Venhoff, N; Lorenzetti, R; Warnatz, K; Unger, S; Seidl, M; Mielenz, D; Schneider, P; Ehl, S; Rensing-Ehl, A; Smulski, CR; Rizzi, M
Non-apoptotic FAS signaling controls mTOR activation and extrafollicular maturation in human B cells.
SCI IMMUNOL. 2024; 9(91): eadj5948
Doi: 10.1126/sciimmunol.adj5948
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** Thiel, J; Schmidt, FM; Lorenzetti, R; Troilo, A; Janowska, I; Nießen, L; Pfeiffer, S; Staniek, J; Benassini, B; Bott, MT; Korzhenevich, J; Konstantinidis, L; Burgbacher, F; Dufner, AK; Frede, N; Voll, RE; Stuchly, J; Bakardjieva, M; Kalina, T; Smulski, CR; Venhoff, N; Rizzi, M
Defects in B-lymphopoiesis and B-cell maturation underlie prolonged B-cell depletion in ANCA-associated vasculitis.
Ann Rheum Dis. 2024;
Doi: 10.1136/ard-2024-225587
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** Benassini, B; Staniek, J; Schmidt, F; Lorenzetti, R; Smulski, C; Schneider, P; Thiel, J; Venhoff, N; Rizzi, M
Dysregulation of B cell function in ANCA-associated vasculitis
EUR J IMMUNOL. 2023; 53: 391-391.
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** Lorenzetti, R; Staniek, J; Rizzi, M; Voll, R; Venhoff, N; Thiel, J
TET2 MOSAICISM IN HUMAN IS ASSOCIATED WITH A COMPLEX PHENOTYPE INCLUDING LYMPHOPROLIFERATION, AUTOIMMUNITY, IMMUNODEFICIENCY, AND HEMATOLOGIC MALIGNANCY
ANN RHEUM DIS. 2023; 82: 807-808.
Doi: 10.1136/annrheumdis-2023-eular.3487
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